Changes in AA-GSH cycle activity following Botrytis cinerea infection were studied in tomato whole-leaf extracts as well as in chloroplasts, mitochondria, and peroxisomes.
First-pass extracted concept
ascorbate-glutathione cycle
Candidate: concept label1 source documents6 linked claims
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AA-GSH cycle
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Supporting Sources
Linked Claims
The oxidative effect of Botrytis cinerea infection affects all examined cellular compartments, with mitochondria and peroxisomes showing the most pronounced changes.
Quoted textsource-backed
The oxidative effect of infection affected all cellular compartments although mitochondria and peroxisomes underwent the most pronounced changes.
Changes in ascorbate-glutathione cycle activity may be partly related to Botrytis cinerea-induced promotion of senescence that favors disease progress.
Quoted textsource-backed
The changes in the AA-GSH cycle activity could partly be related to the B. cinerea-induced promotion of senescence that favoured disease progress.
Botrytis cinerea breaks down the protective antioxidant barrier of the ascorbate-glutathione cycle at both cellular and organellar levels.
Quoted textsource-backed
It was concluded that B. cinerea was able to break down the protective antioxidant barrier of the AA-GSH cycle at both the cellular and organellar levels.
The oxidative shift is associated with decreased concentrations and redox ratios of ascorbate and glutathione pools and insufficient MDHAR, DHAR, and GR activity for antioxidant regeneration.
Quoted textsource-backed
It was manifested by the significant decline in concentrations and redox ratios of the ascorbate and glutathione pools as well as by the insufficient activity of MDHAR, DHAR, and GR needed for antioxidant regeneration.
Botrytis cinerea infection alters ascorbate-glutathione cycle activity in tomato leaves and in chloroplasts, mitochondria, and peroxisomes.
Quoted textsource-backed
Changes in AA-GSH cycle activity following Botrytis cinerea infection were studied in tomato whole-leaf extracts as well as in chloroplasts, mitochondria, and peroxisomes.
Botrytis cinerea infection shifts the cellular redox balance of tomato leaves toward a more oxidative state.
Quoted textsource-backed
Apart from organelle-specific variations, a general shift of the cellular redox balance towards the oxidative state was found.