First-pass extracted concept

astrocytic Ca2+ signalling

Candidate: concept label1 source documents5 linked claims
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Evidence Snippets

Central to this paradigm shift is the role of astrocytic Ca²⁺ signalling in modulating synaptic activity, plasticity, and network behaviour.
Evidence 1Source 1DOIPubMedprovenance

Supporting Sources

Linked Claims

Claim 1disease associationsupports2025Source 1DOIPubMed

Astrocytic Ca2+ waves are implicated in pathological states such as epilepsy.

Quoted textsource-backed
These discoveries extended to network-level phenomena, implicating astrocytic Ca2+ waves in pathological states like epilepsy.
Claim 2functional rolesupports2025Source 1DOIPubMed

Astrocytes actively participate in synaptic communication through Ca2+-dependent molecular signalling referred to as gliotransmission.

Quoted textsource-backed
Astrocytes, once considered passive support cells, have emerged as active participants in synaptic communication through Ca2+-dependent molecular signalling often referred to as gliotransmission.
Claim 3mechanismsupports2025Source 1DOIPubMed

Astrocytic Ca2+ signalling modulates synaptic activity, plasticity, and network behaviour.

Quoted textsource-backed
Central to this paradigm shift is the role of astrocytic Ca²⁺ signalling in modulating synaptic activity, plasticity, and network behaviour.
Claim 4mechanismsupports2025Source 1DOIPubMed

Intracellular Ca2+ fluctuations in astrocytes trigger release of signalling molecules that influence excitatory and inhibitory neuronal circuits.

Quoted textsource-backed
Carmignoto's research demonstrated that intracellular Ca2+ fluctuations in astrocytes trigger the release of signalling molecules, influencing both excitatory and inhibitory neuronal circuits.
Claim 5therapeutic implicationsupports2025Source 1DOIPubMed

Astrocytic dysfunction is related to epilepsy and dopaminergic dysregulation, suggesting therapeutic avenues through astrocyte-specific interventions.

Quoted textsource-backed
In the context of pathology, Carmignoto's studies related astrocytic dysfunction to epilepsy and dopaminergic dysregulation, suggesting new therapeutic avenues through astrocyte-specific interventions.