First-pass extracted concept

BMPR-1B

Candidate: concept label1 source documents4 linked claims
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Evidence Snippets

Our study was undertaken to specify the function of BMPR-1B in steroidogenic enzyme gene expression, testosterone production and reproductive development in male mice.
Evidence 1Source 1DOIPubMedprovenance

Supporting Sources

Linked Claims

Claim 1context dependent effectsupports2023Source 1DOIPubMed

In adult testicular culture and seminiferous tubule conditioned Leydig cell culture, anti-BMPR-1B reduced testosterone, whereas in normal and Leydig cell conditioned Leydig cell culture it increased testosterone.

Quoted textsource-backed
In adult testicular culture and seminiferous tubule conditioned Leydig cell culture, anti-BMPR-1B reduced testosterone, while in normal and Leydig cell conditioned Leydig cell culture it increased testosterone levels.
Claim 2developmental effectsupports2023Source 1DOIPubMed

In pubertal mice, anti-BMPR-1B reduced gonadotrophin-stimulated seminal vesicle growth.

Quoted textsource-backed
In pubertal mice, anti-BMPR-1B reduced gonadotrophin stimulated seminal vesicle growth.
Claim 3functional rolesupports2023Source 1DOIPubMed

BMPR-1B modulates steroidogenic enzyme gene expression, testosterone production, and reproductive development in male mouse testis.

Quoted textsource-backed
Our study was undertaken to specify the function of BMPR-1B in steroidogenic enzyme gene expression, testosterone production and reproductive development in male mice... In conclusion, BMPR-1B has specific developmental functions in the autocrine and paracrine regulation of testicular steroidogenic enzyme gene expression and testosterone production in adults and in the development of seminal vesicles during puberty.
Claim 4perturbation effectsupports2023Source 1DOIPubMed

In adult mice, anti-BMPR-1B increased testosterone and Hsd3b1 mRNA and decreased Hsd3b6 and Cyp19 mRNA.

Quoted textsource-backed
In adult mice, anti-BMPR-1B increased testosterone and Hsd3b1 but decreased Hsd3b6 and Cyp19 mRNA.