FsK is presented as an endophytic fungus that modulates tomato root development and hormone pathways during early interaction.
First-pass extracted concept
Fusarium solani strain FsK
Aliases
FsK
Extracted Explainers
Evidence Snippets
Supporting Sources
Linked Claims
The reported coordinated fungal-plant hormone control supports the potential of FsK as a tomato biostimulant.
These results reveal coordinated fungal-plant hormone control at the transcriptional level that promotes root development during early interaction and support FsK's potential as a biostimulant.
FsK curtails tomato leaf ethylene biosynthesis through down-regulation of ACC oxidase genes with isoform-specific changes in ACC synthase and transient early expression of ETR1.
ET biosynthesis was curtailed in leaves via down-regulation of ACC oxidase (ACO1-3), with isoform-specific changes in ACC synthase (ACS). The ET receptor ETR1 was transiently expressed early (T1-T2).
FsK shows staged activation of fungal ethylene-biosynthesis genes during early interaction with tomato.
FsK itself showed staged activation of fungal ET-biosynthesis genes.
FsK broadly affects tomato leaf ABA biosynthetic and homeostasis gene expression in a pattern interpreted as reduced de novo ABA synthesis with enhanced deconjugation of stored ABA.
In leaves, FsK broadly affected ABA biosynthetic and homeostasis genes (ZEP1, NCED1, ABA2, AAO1, ABA-GT, BG1), indicating reduced de novo synthesis with enhanced deconjugation of stored ABA.
Fusarium solani strain FsK alters tomato root system architecture by increasing root area, lateral root number, root-hair length, and fresh biomass during in vitro co-cultivation.
FsK altered root system architecture, increasing root area, lateral root number, root-hair length, and fresh biomass.