First-pass extracted concept

IAA3

Candidate: concept label1 source documents6 linked claims

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Aliases

SHY2/IAA3

Evidence Snippets

The gain-of-function mutation in IAA3 caused hyposensitivity to light, whereas disruption of IAA3 led to an elongated hypocotyl under different light intensity conditions.

Evidence 1Source 1DOI PubMedprovenance

Supporting Sources

Source 1primary paper2021PLoS GeneticsDOI PubMed

1 evidence snippet 6 linked claims Claim 1 Claim 2 Claim 3

Linked Claims

Claim 1gene regulationsupports2021Source 1DOI PubMed

IAA3 negatively regulates expression of PIF-dependent genes.

Claim 2genetic dependencysupports2021Source 1DOI PubMed

The function of IAA3 in hypocotyl elongation depends on PIFs.

Claim 3mechanism of actionsupports2021Source 1DOI PubMed

IAA3 attenuates the DNA-binding activities of PIFs to target genes.

Claim 4mechanistic linksupports2021Source 1DOI PubMed

IAA3 mediates a direct link between light signaling and auxin signaling through PIF transcription factors in Arabidopsis.

Claim 5phenotype requirementsupports2021Source 1DOI PubMed

IAA3 is required for light-regulated hypocotyl growth in Arabidopsis.

Claim 6physical interactionsupports2021Source 1DOI PubMed

IAA3 interacts with PIFs in vitro and in vivo.