First-pass extracted concept

innate kinin pathway

Candidate: concept label1 source documents4 linked claims
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Evidence Snippets

Here, we review knowledge emerging from investigations of the role of the kinin system in the mechanisms that link innate to the adaptive phase of immunity.
Evidence 1Source 1DOIPubMedprovenance

Supporting Sources

Linked Claims

Claim 1mechanistic summarysupports2008Source 1DOIPubMed

Bradykinin acts as an endogenous danger signal that induces dendritic cell maturation through bradykinin B2 receptors.

Quoted textsource-backed
bradykinin is an endogenous danger signal that induces dendritic cell (DC) maturation via G protein-coupled bradykinin B2 receptors (B2R)
Claim 2mechanistic summarysupports2008Source 1DOIPubMed

Failure of dendritic cells from B2R-deficient mice to sense parasite-released kinin danger signals is associated with lower frequencies of interferon-gamma-producing effector T cells.

Quoted textsource-backed
DCs from B2R-/- mice (susceptible phenotype) failed to sense kinin 'danger' signals proteolytically released by parasites, explaining why these mutant mice display lower frequencies of interferon-gamma-producing effector T-cells
Claim 3mechanistic summarysupports2008Source 1DOIPubMed

In mouse models of Trypanosoma cruzi infection, kinins have an immunostimulatory role.

Quoted textsource-backed
The immunostimulatory role of kinins was recently confirmed in two different mouse models of Trypanosoma cruzi infection
Claim 4mechanistic summarysupports2008Source 1DOIPubMed

The balance between cruzipain-mediated kinin generation and angiotensin-converting enzyme-mediated kinin degradation governs dendritic cell maturation and TH1 development through a B2R-dependent innate pathway.

Quoted textsource-backed
the balance between cruzipain and angiotensin-converting enzyme, respectively acting as kinin-generating and degrading enzymes, governs extent of DC maturation and TH1 development via the B2R-dependent innate pathway