First-pass extracted concept

neurocircuitry mapping

Candidate: concept label1 source documents4 linked claims
Live refresh every 5sNext refresh in 5s

Evidence Snippets

Research in this dynamically developing field has progressed rapidly due to techniques allowing targeted transgenesis and neurocircuitry mapping
Evidence 1Source 1DOIPubMedprovenance

Supporting Sources

Linked Claims

Claim 1mechanistic scopesupports2017Source 1DOIPubMed

Brain control of peripheral glucose metabolism can occur by modulating hepatic gluconeogenesis and by regulating glucose uptake into brown adipose tissue.

Quoted textsource-backed
The brain controls peripheral glucose metabolism, for example by modulating hepatic gluconeogenesis or by regulating glucose uptake into brown adipose tissue.
Claim 2methodological enablersupports2017Source 1DOIPubMed

Targeted transgenesis and neurocircuitry mapping enabled rapid progress in defining responsive neurons, associated molecular mechanisms, and downstream neurocircuits involved in CNS control of peripheral glucose metabolism.

Quoted textsource-backed
Research in this dynamically developing field has progressed rapidly due to techniques allowing targeted transgenesis and neurocircuitry mapping, which have defined the primary responsive neurons, associated molecular mechanisms and downstream neurocircuitries and processes involved.
Claim 3physiological scopesupports2017Source 1DOIPubMed

CNS control of peripheral glucose metabolism includes regulation of liver, brown adipose tissue, and pancreatic function.

Quoted textsource-backed
Here we review the brain regions, neurons and molecular mechanisms by which the CNS controls peripheral glucose metabolism, particularly via regulation of liver, brown adipose tissue and pancreatic function
Claim 4review scope summarysupports2017Source 1DOIPubMed

The central nervous system has an important role in regulating peripheral insulin sensitivity and glucose homeostasis.

Quoted textsource-backed
The central nervous system (CNS) has an important role in the regulation of peripheral insulin sensitivity and glucose homeostasis.