The functional role of the CeA in feeding regulation was examined using chemogenetic and optogenetic activation.
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optogenetic activation
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Further, we demonstrate that optogenetic activation of CIII md neurons evokes calcium increases in these neurons.
The abstract reports optogenetic activation of GABAergic LPBN neurons and optogenetic activation of glutamatergic LPBN neurons.
We show that optogenetic activation of these neurons is sufficient to induce CTA in the absence of anorexigenic substances
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Optogenetic activation of CIII md neurons evokes calcium increases in these neurons.
Co-activation of CIII md neurons with additional cell types facilitates larval contraction responses.
Optogenetic activation of GABAergic LPBN neurons alleviates neuropathic pain-like behavior without affecting basal nociception.
Optogenetic activation of glutamatergic LPBN neurons or inhibition of GABAergic LPBN neurons induces neuropathic pain-like behavior in naive mice.
Optogenetic activation of PBel CGRP neurons is sufficient to induce conditioned taste aversion in mice in the absence of anorexigenic substances.
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We show that optogenetic activation of these neurons is sufficient to induce CTA in the absence of anorexigenic substances