Current evidence has indicated that exposure to passive smoking can lead to a 70-80% increase in the risk of coronary heart disease, nearly as much as light smoking.
First-pass extracted concept
passive smoking
Candidate: concept label1 source documents4 linked claims
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Aliases
passive smoke, secondhand smoke, SHS
Evidence Snippets
Supporting Sources
Linked Claims
The review concludes that the combined effects of passive smoke exposure support a causal relationship with cardiovascular disease development.
Quoted textsource-backed
Acting alone or in synergy, the above mentioned effects suggest a causal relationship between exposure to passive smoking and the development of cardiovascular disease.
The review argues that passive smoking may induce inflammatory responses that contribute to cardiovascular disease development.
Quoted textsource-backed
In this review we present the different mechanisms through which passive smoking may induce an inflammatory response that may lead to the development of cardiovascular disease.
The review summarizes that passive smoke can induce vascular damage including endothelial inflammation, atherosclerosis development, lipid peroxidation, cytokine and acute phase protein alterations, and platelet aggravation.
Quoted textsource-backed
Passive smoke itself, is a volatile mixture of numerous toxins, chemicals and carcinogens, that interact with in vivo mechanisms and induce vascular damage, including endothelium inflammation, atherosclerosis development, lipid peroxidisation, alterations in cytokines and acute phase proteins (such as CRP), as well as platelet aggravation.
The review states that passive smoking exposure is associated with a 70-80% increase in coronary heart disease risk.
Quoted textsource-backed
Current evidence has indicated that exposure to passive smoking can lead to a 70-80% increase in the risk of coronary heart disease, nearly as much as light smoking.