ROS signaling is described as an integrated component of CDPK-mediated abiotic stress responses, including antioxidant defense regulation.
First-pass extracted concept
reactive oxygen species signaling
Aliases
ROS, ROS signaling
Extracted Explainers
Evidence Snippets
the review explores the critical cross-talk between CDPKs signaling networks and phytohormone pathways, particularly abscisic acid (ABA), and their integration with reactive oxygen species (ROS) and MAPK signaling cascades
Moreover, the plant stress tolerance is suggested to be influenced by stress-induced signalling mechanisms, which are mediated by reactive oxygen species (ROS).
ROS (reactive oxygen species) cause severe cellular damage by peroxidation and de-esterification of membrane-lipids, however, current models also define a pivotal signaling function of ROS in triggering tolerance against stress.
Supporting Sources
Linked Claims
Plant stress tolerance is influenced by stress-induced signaling mechanisms mediated by reactive oxygen species.
CDPKs orchestrate plant responses to abiotic stress through substrate phosphorylation, ROS regulation, kinase cross-talk, and precise spatiotemporal activity.
through substrate phosphorylation, ROS regulation, kinase cross-talk, and precise spatiotemporal activity, playing a central role in orchestrating plant responses to abiotic stress
CDPK signaling networks show critical cross-talk with ABA pathways and integrate with ROS and MAPK signaling cascades.
the critical cross-talk between CDPKs signaling networks and phytohormone pathways, particularly abscisic acid (ABA), and their integration with reactive oxygen species (ROS) and MAPK signaling cascades
ROS participate in crosstalk with stress-triggered hormonal signaling including the abscisic acid pathway and endogenous redox and metabolite signals.
ROS have both damaging cellular effects and a pivotal signaling function in triggering stress tolerance.
Abiotic stress-responsive signaling networks are reviewed in the context of ROS, lipid-derived signals, and stomatal signaling.
Environmental stress effects converge on ionic and osmotic imbalance, impaired photosynthesis, cellular energy depletion, and redox imbalance.