Together, these results demonstrate that SDR7-6 functions as a previously unrecognized downstream component of phyB-mediated red-light signaling and is involved in red-light-dependent lesion formation in rice.
First-pass extracted concept
SDR7-6
Aliases
sdr7-6
Evidence Snippets
Supporting Sources
Linked Claims
Knockout of phyB markedly alleviates lesion development in sdr7-6, supporting a role for phyB-mediated red-light signaling in the lesion phenotype.
Knockout of <i>phyB</i> markedly alleviated lesion development, confirming the role of phyB-mediated red-light signaling in this process.
SDR7-6 functions as a previously unrecognized downstream component of phyB-mediated red-light signaling involved in red-light-dependent lesion formation in rice.
Together, these results demonstrate that SDR7-6 functions as a previously unrecognized downstream component of phyB-mediated red-light signaling and is involved in red-light-dependent lesion formation in rice
In the rice mutant sdr7-6, lesion formation is exclusively triggered by red light and is independent of light intensity or photoperiod.
We found that lesion formation was exclusively triggered by red light and occurred independently of light intensity or photoperiod.
The sdr7-6 mutant shows significantly reduced photosynthetic capacity, primarily due to disrupted chloroplast ultrastructure.
Physiological analyses revealed a significant reduction in photosynthetic capacity in <i>sdr7-6</i>, primarily due to disrupted chloroplast ultrastructure.
Transcriptome profiling in sdr7-6 indicates strong downregulation of genes associated with chloroplast function, photosynthesis, and light responses.
Consistent with these findings, transcriptome profiling indicated strong downregulation of genes associated with chloroplast function, photosynthesis, and light responses.