Recent research has also shed light on the critical role of thrombus formation, which prevents the dissemination of microorganisms.
First-pass extracted concept
thrombus formation during sepsis
Candidate: concept label1 source documents4 linked claims
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Linked Claims
Histones, HMGB1, microparticles, and secreted granule proteins are described as important contributors to clot formation in sepsis.
Quoted textsource-backed
In addition to the cellular components, alarmins such as histones and high-mobility group box 1, microparticles and secreted granule proteins are all important for clot formation.
Inflammatory responses and coagulation activation cooperate in a complex and synchronous manner during host defense against infection.
Quoted textsource-backed
The inflammatory response and the activation of coagulation are two important responses in a host's defense against infection. These mechanisms do not work independently, but cooperate in a complex and synchronous manner.
Leukocytes, platelets, erythrocytes, and vascular endothelial cells are described as significant cellular contributors to thrombus development together with coagulation activation in sepsis.
Quoted textsource-backed
The cellular components of blood vessels, i.e. leukocytes, platelets, erythrocytes, and vascular endothelial cells, play significant roles in the development of thrombi in combination with activation of the coagulation system.
Thrombus formation is described as playing a critical role in preventing dissemination of microorganisms during sepsis.
Quoted textsource-backed
Recent research has also shed light on the critical role of thrombus formation, which prevents the dissemination of microorganisms.