First-pass extracted concept

64PHR

Candidate: toolkit item1 source documents5 linked claims

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Aliases

z64Phr

Evidence Snippets

light induction of 64PHR, a DNA repair enzyme

Evidence 1Source 1DOI PubMedprovenance

Supporting Sources

Source 1primary paper2009Cell CycleDOI PubMed

1 evidence snippet 5 linked claims Claim 1 Claim 2 Claim 3

Linked Claims

Claim 1biological activitysupports2009Source 1DOI PubMed

Light-dependent UV tolerance is a cell-autonomous phenomenon in zebrafish.

Quoted textsource-backed

Here we show that light-dependent UV-tolerance is a cell autonomous phenomenon in zebrafish.

Claim 2causal requirementsupports2009Source 1DOI PubMed

Light induction of 64PHR and the subsequent 64PHR-mediated light-dependent DNA repair are prerequisites for light-mediated UV tolerance.

Quoted textsource-backed

light induction of 64PHR, a DNA repair enzyme, and the subsequent light-dependent DNA repair mediated by this enzyme are prerequisites for light-mediated UV tolerance

Claim 3gene expression controlsupports2009Source 1DOI PubMed

Light-induced activation of MAPK signaling pathways controls expression of z64Phr and zCry1a.

Quoted textsource-backed

light-induced activation of these pathways controls the expression of two evolutionary-related genes, z64Phr and zCry1a

Claim 4shared regulationsupports2009Source 1DOI PubMed

Light-dependent DNA repair and circadian clock entrainment share common regulatory pathways in zebrafish.

Quoted textsource-backed

revealing that light-dependent DNA repair and the entrainment of circadian clock share common regulatory pathways

Claim 5structural relationshipsupports2009Source 1DOI PubMed

64PHR is evolutionarily related to and has high structural similarity to animal CRY.

Quoted textsource-backed

64PHR is evolutionary related to and has a high degree of structural similarity to animal CRY