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CPK6

Candidate: toolkit item1 source documents16 linked claims
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Aliases

Calcium-Dependent Protein Kinase CPK6

Evidence Snippets

A Ca(2+)-dependent protein kinase, CPK6
Evidence 1Source 1DOIPubMedprovenance

Supporting Sources

Linked Claims

Claim 1biological functionsupports2013Source 1DOIPubMed

CPK6 positively functions in yeast elicitor-induced stomatal closure in Arabidopsis.

Quoted textsource-backed
Disruption of CPK6 gene impaired induction by YEL of stomatal closure
Claim 2biological functionsupports2013Source 1DOIPubMed

CPK6 positively functions in yeast elicitor-mediated inhibition of light-induced stomatal opening in Arabidopsis.

Quoted textsource-backed
Disruption of CPK6 gene impaired induction by YEL of stomatal closure and inhibition by YEL of light-induced stomatal opening.
Claim 3calcium signaling rolesupports2013Source 1DOIPubMed

CPK6 contributes to transient yeast elicitor-induced elevations in cytosolic free Ca2+ concentration in guard cells.

Quoted textsource-backed
transient elevations elicited by YEL in cytosolic-free Ca(2+) concentration were suppressed in cpk6-2 and cpk6-1 guard cells
Claim 4channel regulationsupports2013Source 1DOIPubMed

CPK6 is required for full activation by yeast elicitor of nonselective Ca2+-permeable cation channels in guard cells.

Quoted textsource-backed
Activation by YEL of nonselective Ca(2+)-permeable cation channels was impaired in cpk6-2 guard cells
Claim 5channel regulationsupports2013Source 1DOIPubMed

CPK6 is required for yeast elicitor activation of slow anion channels in guard cells.

Quoted textsource-backed
YEL activated slow anion channels in wild-type guard cells but not in cpk6-2 or cpk6-1
Claim 6channel regulationsupports2013Source 1DOIPubMed

CPK6 is required for yeast elicitor inhibition of inward-rectifying K+ channels in guard cells.

Quoted textsource-backed
YEL inhibited inward-rectifying K(+) channels in wild-type guard cells but not in cpk6-2 or cpk6-1
Claim 7functional rolesupports2013Source 1DOIPubMed

CPK6 positively functions in yeast elicitor-induced stomatal closure in Arabidopsis.

Quoted textsource-backed
Disruption of CPK6 gene impaired induction by YEL of stomatal closure
Claim 8functional rolesupports2013Source 1DOIPubMed

CPK6 positively functions in yeast elicitor-mediated inhibition of light-induced stomatal opening in Arabidopsis.

Quoted textsource-backed
Disruption of CPK6 gene impaired induction by YEL of stomatal closure and inhibition by YEL of light-induced stomatal opening.
Claim 9mechanistic effectsupports2013Source 1DOIPubMed

CPK6 contributes to transient yeast elicitor-induced elevations in cytosolic free Ca2+ concentration in guard cells.

Quoted textsource-backed
transient elevations elicited by YEL in cytosolic-free Ca(2+) concentration were suppressed in cpk6-2 and cpk6-1 guard cells
Claim 10mechanistic effectsupports2013Source 1DOIPubMed

CPK6 is required for full activation by yeast elicitor of nonselective Ca2+-permeable cation channels in guard cells.

Quoted textsource-backed
Activation by YEL of nonselective Ca(2+)-permeable cation channels was impaired in cpk6-2 guard cells
Claim 11mechanistic effectsupports2013Source 1DOIPubMed

CPK6 is required for yeast elicitor activation of slow anion channels in guard cells.

Quoted textsource-backed
YEL activated slow anion channels in wild-type guard cells but not in cpk6-2 or cpk6-1
Claim 12mechanistic effectmixed2013Source 1DOIPubMed

CPK6 is required for yeast elicitor-induced hydrogen peroxide accumulation in guard cells and in the apoplast of rosette leaves, but not for yeast elicitor-induced hydrogen peroxide production in the apoplast of rosette leaves.

Quoted textsource-backed
The cpk6-2 and cpk6-1 mutations inhibited YEL-induced hydrogen peroxide accumulation in guard cells and apoplast of rosette leaves but did not affect YEL-induced hydrogen peroxide production in the apoplast of rosette leaves.
Claim 13mechanistic effectsupports2013Source 1DOIPubMed

CPK6 is required for yeast elicitor inhibition of inward-rectifying K+ channels in guard cells.

Quoted textsource-backed
YEL inhibited inward-rectifying K(+) channels in wild-type guard cells but not in cpk6-2 or cpk6-1
Claim 14pathway positionsupports2013Source 1DOIPubMed

CPK6 is a convergent point of signaling pathways for stomatal closure in response to abiotic and biotic stress in Arabidopsis.

Quoted textsource-backed
These results suggest that CPK6 positively functions in induction by YEL of stomatal closure and inhibition by YEL of light-induced stomatal opening in Arabidopsis and is a convergent point of signaling pathways for stomatal closure in response to abiotic and biotic stress.
Claim 15pathway rolesupports2013Source 1DOIPubMed

CPK6 is a convergent point of signaling pathways for stomatal closure in response to abiotic and biotic stress.

Quoted textsource-backed
These results suggest that CPK6 positively functions in induction by YEL of stomatal closure and inhibition by YEL of light-induced stomatal opening in Arabidopsis and is a convergent point of signaling pathways for stomatal closure in response to abiotic and biotic stress.
Claim 16reactive oxygen rolesupports2013Source 1DOIPubMed

CPK6 is required for yeast elicitor-induced hydrogen peroxide accumulation in guard cells and in the apoplast of rosette leaves, but not for yeast elicitor-induced hydrogen peroxide production in the apoplast of rosette leaves.

Quoted textsource-backed
The cpk6-2 and cpk6-1 mutations inhibited YEL-induced hydrogen peroxide accumulation in guard cells and apoplast of rosette leaves but did not affect YEL-induced hydrogen peroxide production in the apoplast of rosette leaves.