We used loss-of-function mutants to study three Arabidopsis thaliana sensor histidine kinases, AHK2, AHK3, and CRE1/AHK4, known to be cytokinin receptors.
First-pass extracted concept
CRE1/AHK4
Aliases
AHK4
Evidence Snippets
Supporting Sources
Linked Claims
The cytokinin receptors have partially redundant functions.
Together, the analyses reveal partially redundant functions of the cytokinin receptors
Genetic analysis indicated a cytokinin-dependent endospermal and/or maternal control of embryo size.
Genetic analysis indicated a cytokinin-dependent endospermal and/or maternal control of embryo size.
Increased cytokinin content in receptor mutants indicates homeostatic control of steady-state cytokinin levels through signaling.
Increased cytokinin content of receptor mutants indicates a homeostatic control of steady state cytokinin levels through signaling.
AHK2, AHK3, and CRE1/AHK4 are cytokinin receptors.
AHK2, AHK3, and CRE1/AHK4, known to be cytokinin receptors
Previously reported modulation of red light signaling by A-type response regulators may not depend on cytokinin.
Unchanged red light sensitivity of mutant hypocotyl elongation suggests that previously reported modulation of red light signaling by A-type response regulators may not depend on cytokinin.
Loss-of-function cytokinin receptor mutants show more rapid germination, reduced light requirement, and decreased far-red light sensitivity, indicating cytokinin functions in seed germination control.
Mutant seeds had more rapid germination, reduced requirement for light, and decreased far-red light sensitivity, unraveling cytokinin functions in seed germination control.
Triple cytokinin receptor mutant seeds are more than twice as large as wild-type seeds.
Triple mutant seeds were more than twice as large as wild-type seeds.