First-pass extracted concept

ost1-2

Candidate: toolkit item1 source documents5 linked claims

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Aliases

ost1 mutant

Evidence Snippets

we isolated two allelic Arabidopsis mutants (ost1-1 and ost1-2)

Evidence 1Source 1DOI PubMedprovenance

Supporting Sources

Source 1primary paper2002The Plant CellDOI PubMed

1 evidence snippet 5 linked claims Claim 1 Claim 2 Claim 3

Linked Claims

Claim 1epistasis inferencesupports2002Source 1DOI PubMed

Applied hydrogen peroxide or calcium elicits the same degree of stomatal closure in ost1 mutants as in wild type, suggesting OST1 acts between ABA perception and ROS production.

Quoted textsource-backed

applied H(2)O(2) or calcium elicited the same degree of stomatal closure in ost1 as in the wild type. These results suggest that OST1 acts in the interval between ABA perception and ROS production.

Claim 2genetic perturbation effectsupports2002Source 1DOI PubMed

ost1 mutations disrupt ABA induction of stomatal closure and ABA inhibition of light-induced stomatal opening.

Quoted textsource-backed

These recessive ost1 mutations disrupted ABA induction of stomatal closure as well as ABA inhibition of light-induced stomatal opening.

Claim 3genetic perturbation effectsupports2002Source 1DOI PubMed

Recessive ost1 mutations impair the ability to limit transpiration upon drought in Arabidopsis.

Quoted textsource-backed

we isolated two allelic Arabidopsis mutants (ost1-1 and ost1-2) impaired in the ability to limit their transpiration upon drought

Claim 4pathway specificitysupports2002Source 1DOI PubMed

ost1 mutations do not affect stomatal regulation by light or CO2, supporting a specific role for OST1 in ABA signaling.

Quoted textsource-backed

the ost1 mutations did not affect stomatal regulation by light or CO(2), suggesting that OST1 is involved specifically in ABA signaling

Claim 5signaling pathway positionsupports2002Source 1DOI PubMed

ABA-induced ROS production is disrupted in ost1 guard cells.

Quoted textsource-backed

ABA-induced ROS production was disrupted in ost1 guard cells