Here we characterize a new protein that associates with both circadian clock and photoreceptor components, named PHOTOPERIODIC CONTROL OF HYPOCOTYL1 (PCH1).
First-pass extracted concept
PHOTOPERIODIC CONTROL OF HYPOCOTYL1
Aliases
PCH1
Evidence Snippets
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Linked Claims
PCH1 is a new protein that associates with both circadian clock and photoreceptor components.
Here we characterize a new protein that associates with both circadian clock and photoreceptor components, named PHOTOPERIODIC CONTROL OF HYPOCOTYL1 (PCH1).
PCH1 binds phytochrome B in a red light-dependent manner.
PCH1 peaks at dusk, binds phytochrome B (phyB) in a red light-dependent manner
PCH1 co-localizes with phytochrome B into photobodies.
and co-localizes with phyB into photobodies.
PCH1 regulates photoperiod-responsive growth by integrating the clock with light perception pathways through modulating daily phyB signaling.
Thus, PCH1 is a new factor that regulates photoperiod-responsive growth by integrating the clock with light perception pathways through modulating daily phyB-signaling.
PCH1 is necessary and sufficient to promote the biogenesis of large photobodies and maintain an active phyB pool after light exposure.
PCH1 is necessary and sufficient to promote the biogenesis of large photobodies to maintain an active phyB pool after light exposure
Constitutive expression of PCH1 shortens hypocotyls independent of day length.
while constitutive expression of PCH1 shortens hypocotyls independent of day length.
Loss of PCH1 causes overly elongated hypocotyls specifically under short days.
pch1 seedlings have overly elongated hypocotyls specifically under short days
Manipulating PCH1 alters PIF4 levels and regulates light-responsive gene expression.
Manipulating PCH1 alters PHYTOCHROME INTERACTING FACTOR 4 levels and regulates light-responsive gene expression.
PCH1 potentiates red-light signaling and prolongs memory of prior illumination.
potentiating red-light signaling and prolonging memory of prior illumination.