Toolkit/Ca2+/cAMP response element decoy oligodeoxynucleotide
Ca2+/cAMP response element decoy oligodeoxynucleotide
Also known as: Ca(2+)/cAMP response element (CRE)-decoy ODN, CRE-decoy ODN
Taxonomy: Mechanism Branch / Architecture. Workflows sit above the mechanism and technique branches rather than replacing them.
Summary
The Ca2+/cAMP response element decoy oligodeoxynucleotide (CRE-decoy ODN) is an oligodeoxynucleotide perturbation reagent used in the rat suprachiasmatic nucleus to interfere with CREB-dependent transcription. In the cited study, it blocked glutamate-induced Per1 mRNA accumulation and prevented glutamate-induced phase advances of the circadian clock, implicating CREB-dependent Per1 activation in light-responsive circadian signaling.
Usefulness & Problems
Why this is useful
This reagent is useful for testing whether transcription through a Ca2+/cAMP response element is required in a defined signaling pathway. In the reported rat SCN experiments, it enabled functional disruption of CREB-dependent transcription linking glutamatergic input to Per1 induction and circadian phase shifting.
Problem solved
It addresses the problem of causally perturbing CREB-dependent gene regulation in the suprachiasmatic nucleus without directly altering the CREB protein or the Per1 locus. The cited work used it to determine whether CRE-mediated transcription is necessary for glutamate-triggered Per1 expression and clock phase advance.
Taxonomy & Function
Primary hierarchy
Mechanism Branch
Architecture: A composed arrangement of multiple parts that instantiates one or more mechanisms.
Techniques
No technique tags yet.
Target processes
recombinationsignalingInput: Light
Implementation Constraints
The tool is an oligodeoxynucleotide decoy directed against the Ca2+/cAMP response element and was applied in rat SCN experiments involving glutamatergic stimulation. The supplied evidence does not specify the ODN sequence, chemical modifications, delivery method, or whether exogenous cofactors were required.
The evidence provided comes from a single cited study in rat suprachiasmatic nucleus tissue, so validation breadth is limited. The supplied evidence does not report sequence design, delivery conditions, off-target assessment, kinetics, dose response, or performance in other cell types or organisms.
Validation
Observations
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Inferred from claim c3 during normalization. CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus. Derived from claim c3. Section: abstract. Quoted text: Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
Supporting Sources
Ranked Claims
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
Antisense oligodeoxynucleotide against Per1 blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
Approval Evidence
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
CRE-decoy oligodeoxynucleotide blocks glutamate-induced accumulation of Per1 mRNA.
Furthermore, the CRE-decoy blocked GLU-induced accumulation of Per1 mRNA.
Source:
CREB and Per1 are integral components of the pathway that transduces light-stimulated glutamatergic neurotransmission into phase advance of the circadian clock.
Thus, Ca(2+)/cAMP response element-binding protein (CREB) and Per1 are integral components of the pathway transducing light-stimulated GLU neurotransmission into phase advance of the circadian clock.
Source:
CRE-decoy oligodeoxynucleotide blocks glutamate-induced phase advances in the rat suprachiasmatic nucleus.
Glutamate (GLU)-induced phase advances in the rat SCN were blocked by antisense oligodeoxynucleotide (ODN) against Per1 and Ca(2+)/cAMP response element (CRE)-decoy ODN.
Source:
CRE-decoy oligodeoxynucleotide blocks light-induced phase advances in vivo.
CRE-decoy ODN also blocked light-induced phase advances in vivo.
Source:
CREB-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Ca2+/cAMP Response Element-binding Protein (CREB)-dependent Activation of Per1 Is Required for Light-induced Signaling in the Suprachiasmatic Nucleus Circadian Clock
Source:
Comparisons
Source-backed strengths
The reported strength is pathway-level functional specificity: the CRE-decoy ODN blocked glutamate-induced Per1 mRNA accumulation and blocked glutamate-induced phase advances in the rat SCN. The same study also placed CREB and Per1 as integral components of the pathway transducing light-stimulated glutamatergic neurotransmission into circadian phase advance.
Ranked Citations
- 1.