Source 1review2014Epilepsia
Toolkit/juxtacellular recording
juxtacellular recording
Taxonomy: Technique Branch / Method. Workflows sit above the mechanism and technique branches rather than replacing them.
Summary
Juxtacellular recording is an electrophysiological assay method identified as an emerging technique that can be combined with the optogenetic toolbox. In the cited epilepsy context, it is positioned as a recording approach used alongside light-driven perturbation to help identify cortical and hippocampal neuron subtypes altered in epileptic networks.
Usefulness & Problems
Why this is useful
This method is useful because it links electrophysiological recording with optogenetic manipulation in epilepsy models. The cited source presents this combination as a way to gain insight into neuronal network organization, synchronization, and cell-type-specific alterations in epileptic circuits.
Problem solved
It helps address the problem of identifying which specific cortical and hippocampal neuron subtypes are altered within epileptic networks while those networks are being probed with optogenetic tools. The available evidence supports this role at the level of combined recording and light-based perturbation, but does not provide a more detailed assay specification.
Problem links
Need precise spatiotemporal control with light input
DerivedJuxtacellular recording is an electrophysiological assay method described as an emerging technique that can be combined with the optogenetic toolbox. In the cited epilepsy context, it is used alongside light-based perturbation to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
Taxonomy & Function
Primary hierarchy
Technique Branch
Method: A concrete measurement method used to characterize an engineered system.
Mechanisms
combination with optogenetic light-driven perturbationcombination with optogenetic light-driven perturbationelectrophysiological recordingelectrophysiological recordingTechniques
Functional AssayTarget processes
No target processes tagged yet.
Input: Light
Implementation Constraints
cofactor dependency: cofactor requirement unknownencoding mode: genetically encodedimplementation constraint: context specific validationimplementation constraint: spectral hardware requirementoperating role: sensor
The method is described only as an electrophysiological recording technique that can be combined with optogenetic light stimulation. The source literature mentions use in the context of opsins such as channelrhodopsin-2, halorhodopsin, archaerhodopsin-3, and light-activated Gq-coupled opsins, but the evidence does not specify juxtacellular electrode configuration, preparation type, or construct design requirements.
The supplied evidence is sparse and does not report quantitative performance metrics, recording yield, temporal resolution, or comparative benchmarking against other electrophysiological methods. Independent validation details, specific protocols, and direct experimental outcomes for juxtacellular recording itself are not provided in the extracted evidence.
Validation
Cell-freeBacteriaMammalianMouseHumanTherapeuticIndep. Replication
Supporting Sources
Ranked Claims
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Light-activated Gq protein-coupled opsins are described as a way to selectively activate astrocytes to explore the influence of gliotransmission on epileptic network function.
The influence of gliotransmission on epileptic network function is another topic of great interest that can be further explored by using light-activated Gq protein-coupled opsins for selective activation of astrocytes.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
Channelrhodopsin-2, halorhodopsin, and archaerhodopsin-3 are presented as potent optogenetic candidates for controlling neuronal firing in models of epilepsy and for providing insights into neuronal network organization and synchronization.
The versatility and the electrophysiologic characteristics of the light-sensitive ion-channels channelrhodopsin-2 (ChR2), halorhodopsin (NpHR), and the light-sensitive proton pump archaerhodopsin-3 (Arch) make these optogenetic tools potent candidates in controlling neuronal firing in models of epilepsy and in providing insights into the physiology and pathology of neuronal network organization and synchronization.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Optogenetic tools allow rapid and reversible suppression of epileptic EEG activity upon photoactivation.
Finally, optogenetic tools allow rapid and reversible suppression of epileptic electroencephalography (EEG) activity upon photoactivation.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, including interneuron subclasses, to study activity patterns in distinct brain states and dissect roles in synchrony and seizures.
Opsins allow selective activation of excitatory neurons and inhibitory interneurons, or subclasses of interneurons, to study their activity patterns in distinct brain-states in vivo and to dissect their role in generation of synchrony and seizures.
Approval Evidence
1 source1 linked approval claimfirst-pass slug juxtacellular-recording
The abstract names juxtacellular recording as an emerging technique that can be combined with the optogenetic toolbox.
Source:
combinatorial method usesupports
The optogenetic toolbox can be combined with juxtacellular recording and two-photon guided whole-cell recording to help identify specific cortical and hippocampal neuron subtypes altered in epileptic networks.
The ever-growing optogenetic toolbox can also be combined with emerging techniques that have greatly expanded our ability to record specific subtypes of cortical and hippocampal neurons in awake behaving animals such as juxtacellular recording and two-photon guided whole-cell recording, to identify the specific subtypes of neurons that are altered in epileptic networks.
Source:
Comparisons
Source-backed strengths
A key strength is compatibility with the optogenetic toolbox, allowing electrophysiological measurements to be paired with light-based control of neural activity. The cited review frames this combined use as informative for studying epilepsy-related network function and synchronization.
Compared with native green gel system
juxtacellular recording and native green gel system address a similar problem space.
Shared frame: same top-level item type; same primary input modality: light
Compared with open-source microplate reader
juxtacellular recording and open-source microplate reader address a similar problem space.
Shared frame: same top-level item type; same primary input modality: light
Compared with plant transcriptome profiling
juxtacellular recording and plant transcriptome profiling address a similar problem space.
Shared frame: same top-level item type; same primary input modality: light
Ranked Citations
- 1.